Treating Keen and Persistent Pains Using Laser
Low-level laser therapy (LLLT) is treatment method using low powered lasers that lead to change the cellular performance. LLLT method is widely used as an auxiliary method to cure the skeletal-muscular disorders. In fact, the color laser pulse does not thermally affect the tissue and cause optical reactions in the tissue via optical stimulation. the reciprocal effect of light and tissue (Photobiostimulation) has anti-pain and anti-inflammation effects and will accelerate the tissue restoration. It is proved that the LLLT method not only effective in treating intense pains regarding arthritis, but also for persistent pains.
Treating intense and persistent pains
The clinical usages of radiating laser for treating intense and persistent pains have been consolidated as a common method. This method has been approved by the FDA and US health & human services. laser therapy is very effective in treating a variety of intense and persistent syndromes of which, pain is the main symptom.
Intense traumas accompany a degree of soft tissue damage including inflammation, hematoma, pain, mobility reduction. Sports injuries and incidents also include damage to the muscles, ligaments and joints’ tendons. Persistent pains might be related to the skeletal-muscular system or be a result of a nerve pain, caused by a particular disease. A high percentage of the patients suffering from persistent pains that are referred to laser therapy and do get treated by laser, have not shown a proper response to the other treatment methods.
Therefore, laser therapy can not only better the intense pains caused by arthritis, but also be used to improve the persistent pains caused by diseases. Moreover, it has been proved that it is more cost effective in comparison to the other treatment methods. Another advantage of this treatment method is that no side effects would occur and also its being noninvasive.
The anti-pain mechanism of laser
Some of the factors that cause the anti-pain effect of the laser can be stated as follows:
- Reduction of building and discharging of the stimulants of the nociceptors (histamine, acetylcholine, etc.)
- Reduction of sensitivity of nociceptors and hence the impulses made by them
- Prevention of building pain stimulants (bradykinin)
- Prevention of building inflammation stimulant (PGE2) which increases the effect of other stimulants on the nociceptors.
- Increasing the lymphatic drainage and thus, decreasing tissue edema which will decrease the pressure on the nerves and cause less nerves stimulation.
- Increasing cortex inhibition mechanisms that increase descending anti-pain impulses on the posterior horn of the spinal cord and inhibition of pain feeling on the cortex surface.